Pages that link to "Q28306393"
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The following pages link to The role of checkpoint kinase 1 in sensitivity to topoisomerase I poisons (Q28306393):
Displaying 45 items.
- A genome-wide camptothecin sensitivity screen identifies a mammalian MMS22L-NFKBIL2 complex required for genomic stability (Q24306107) (← links)
- Mechanistic evaluation of the novel HSP90 inhibitor NVP-AUY922 in adult and pediatric glioblastoma (Q24607424) (← links)
- Implication of checkpoint kinase-dependent up-regulation of ribonucleotide reductase R2 in DNA damage response (Q24650185) (← links)
- Inhibition of Hsp90 acts synergistically with topoisomerase II poisons to increase the apoptotic killing of cells due to an increase in topoisomerase II mediated DNA damage (Q25257535) (← links)
- Trial Watch: Targeting ATM-CHK2 and ATR-CHK1 pathways for anticancer therapy (Q26745686) (← links)
- Advances in the clinical development of heat shock protein 90 (Hsp90) inhibitors in cancers (Q27022514) (← links)
- Tumor suppressor protein kinase Chk2 is a mediator of anoikis of intestinal epithelial cells (Q28582401) (← links)
- Topoisomerase I inhibitors: camptothecins and beyond (Q29614976) (← links)
- Dynamics of p53 and NF-κB regulation in response to DNA damage and identification of target proteins suitable for therapeutic intervention (Q31090728) (← links)
- Beclin 1 and UVRAG confer protection from radiation-induced DNA damage and maintain centrosome stability in colorectal cancer cells (Q33793750) (← links)
- Protein kinase A phosphorylation activates Vpr-induced cell cycle arrest during human immunodeficiency virus type 1 infection (Q33990739) (← links)
- The Mre11 nuclease is critical for the sensitivity of cells to Chk1 inhibition (Q34399650) (← links)
- Lack of Bcl11b tumor suppressor results in vulnerability to DNA replication stress and damages (Q34609906) (← links)
- Riccardin D Exerts Its Antitumor Activity by Inducing DNA Damage in PC-3 Prostate Cancer Cells In Vitro and In Vivo (Q34999154) (← links)
- WRN helicase regulates the ATR-CHK1-induced S-phase checkpoint pathway in response to topoisomerase-I-DNA covalent complexes (Q35630642) (← links)
- Enhanced killing of cancer cells by poly(ADP-ribose) polymerase inhibitors and topoisomerase I inhibitors reflects poisoning of both enzymes. (Q35763155) (← links)
- DNA-damage response network at the crossroads of cell-cycle checkpoints, cellular senescence and apoptosis (Q35815150) (← links)
- Heat shock protein 90 inhibition sensitizes acute myelogenous leukemia cells to cytarabine (Q35847836) (← links)
- Human cells enter mitosis with damaged DNA after treatment with pharmacological concentrations of genotoxic agents (Q36196359) (← links)
- Rad50S alleles of the Mre11 complex: questions answered and questions raised (Q36542150) (← links)
- Repair of topoisomerase I-mediated DNA damage (Q36557533) (← links)
- Potent activity of the Hsp90 inhibitor ganetespib in prostate cancer cells irrespective of androgen receptor status or variant receptor expression (Q36642876) (← links)
- Replication-dependent irreversible topoisomerase 1 poisoning is responsible for FdUMP[10] anti-leukemic activity (Q36862902) (← links)
- Chemotherapy induced DNA damage response: convergence of drugs and pathways (Q36901484) (← links)
- Gene expression time-series analysis of camptothecin effects in U87-MG and DBTRG-05 glioblastoma cell lines. (Q36916118) (← links)
- Cisplatin abrogates the geldanamycin-induced heat shock response (Q36948038) (← links)
- Overcoming S-phase checkpoint-mediated resistance: sequence-dependent synergy of gemcitabine and 7-ethyl-10-hydroxycamptothecin (SN-38) in human carcinoma cell lines (Q36952729) (← links)
- Acquired resistance to 17-allylamino-17-demethoxygeldanamycin (17-AAG, tanespimycin) in glioblastoma cells (Q37120694) (← links)
- A phase 1 dose-escalation study of irinotecan in combination with 17-allylamino-17-demethoxygeldanamycin in patients with solid tumors. (Q37718291) (← links)
- Targeting DNA repair and the cell cycle in glioblastoma (Q37960389) (← links)
- Cell cycle modulatory and apoptotic effects of plant-derived anticancer drugs in clinical use or development (Q38088759) (← links)
- Transcriptome analysis of Aspergillus nidulans exposed to camptothecin-induced DNA damage (Q38499531) (← links)
- Targeting the ATR-CHK1 Axis in Cancer Therapy (Q38817102) (← links)
- The heat shock protein 90 inhibitor SNX5422 has a synergistic activity with histone deacetylase inhibitors in induction of death of anaplastic thyroid carcinoma cells (Q38848546) (← links)
- Prospects for the Use of ATR Inhibitors to Treat Cancer. (Q38974279) (← links)
- Chk2 mediates RITA-induced apoptosis (Q39430270) (← links)
- Growth inhibitory effect and Chk1-dependent signaling involved in G2/M arrest on human gastric cancer cells induced by diallyl disulfide (Q39713463) (← links)
- Defective Mre11-dependent activation of Chk2 by ataxia telangiectasia mutated in colorectal carcinoma cells in response to replication-dependent DNA double strand breaks (Q40244137) (← links)
- Topoisomerase 1 Inhibition Promotes Cyclic GMP-AMP Synthase-Dependent Antiviral Responses. (Q41873973) (← links)
- Mismatch repair system decreases cell survival by stabilizing the tetraploid G1 arrest in response to SN-38. (Q41976774) (← links)
- Role of Protein Linked DNA Breaks in Cancer (Q46243206) (← links)
- Molecular Aspects of Circadian Pharmacology and Relevance for Cancer Chronotherapy (Q47162410) (← links)
- ATR/CHK1 inhibitors and cancer therapy (Q47622968) (← links)
- Survivin antagonizes chemotherapy-induced cell death of colorectal cancer cells. (Q55396539) (← links)
- Topoisomerases and cancer chemotherapy: recent advances and unanswered questions (Q90643414) (← links)