Pages that link to "Q26745490"
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The following pages link to DNA damage tolerance by recombination: Molecular pathways and DNA structures (Q26745490):
Displaying 38 items.
- Choices have consequences: the nexus between DNA repair pathways and genomic instability in cancer (Q28066464) (← links)
- Both RAD5-dependent and independent pathways are involved in DNA damage-associated sister chromatid exchange in budding yeast (Q33768750) (← links)
- Pathways and Mechanisms that Prevent Genome Instability in Saccharomyces cerevisiae (Q33879989) (← links)
- Rescue from replication stress during mitosis (Q34550677) (← links)
- Human PrimPol activity is enhanced by RPA. (Q36342625) (← links)
- The Intra-S Checkpoint Responses to DNA Damage (Q37676255) (← links)
- Human somatic cells deficient for RAD52 are impaired for viral integration and compromised for most aspects of homology-directed repair (Q38761267) (← links)
- PrimPol-Prime Time to Reprime (Q38772940) (← links)
- S-phase checkpoint regulations that preserve replication and chromosome integrity upon dNTP depletion (Q39144822) (← links)
- Molecular Basis for K63-Linked Ubiquitination Processes in Double-Strand DNA Break Repair: A Focus on Kinetics and Dynamics. (Q39355026) (← links)
- Close encounters: Moving along bumps, breaks, and bubbles on expanded trinucleotide tracts (Q39424134) (← links)
- FANCI and FANCD2 have common as well as independent functions during the cellular replication stress response (Q47142336) (← links)
- Lesion Bypass and the Reactivation of Stalled Replication Forks. (Q47235095) (← links)
- Building up and breaking down: mechanisms controlling recombination during replication (Q48096382) (← links)
- Main steps in DNA double-strand break repair: an introduction to homologous recombination and related processes. (Q48256770) (← links)
- Collaboration in the actions of Brh2 with resolving functions during DNA repair and replication stress in Ustilago maydis (Q50136435) (← links)
- Complex repeat structure promotes hyper-amplification and amplicon evolution through rolling-circle replication. (Q52714775) (← links)
- Genome Instability Is Promoted by the Chromatin-Binding Protein Spn1 in (Q57288714) (← links)
- Error-free DNA damage tolerance pathway is facilitated by the Irc5 translocase through cohesin (Q57972039) (← links)
- Chronological Switch from Translesion Synthesis to Homology-Dependent Gap Repair (Q58125939) (← links)
- Mutational signature distribution varies with DNA replication timing and strand asymmetry (Q58756653) (← links)
- Telomeric Recombination Induced by DNA Damage Results in Telomere Extension and Length Heterogeneity (Q58782063) (← links)
- Fanconi Anaemia-Like Mph1 Helicase Backs up Rad54 and Rad5 to Circumvent Replication Stress-Driven Chromosome Bridges (Q59797152) (← links)
- Homologous Recombination: To Fork and Beyond (Q60919487) (← links)
- The Emerging Role of Cohesin in the DNA Damage Response (Q60932044) (← links)
- PDIP38/PolDIP2 controls the DNA damage tolerance pathways by increasing the relative usage of translesion DNA synthesis over template switching (Q64058541) (← links)
- Structure-Specific Endonucleases and the Resolution of Chromosome Underreplication (Q64075900) (← links)
- The Response to DNA Damage at Telomeric Repeats and Its Consequences for Telomere Function (Q64387155) (← links)
- Sml1 Inhibits the DNA Repair Activity of Rev1 in Saccharomyces cerevisiae during Oxidative Stress (Q89473917) (← links)
- From R-Loops to G-Quadruplexes: Emerging New Threats for the Replication Fork (Q89880050) (← links)
- AKT inhibition impairs PCNA ubiquitylation and triggers synthetic lethality in homologous recombination-deficient cells submitted to replication stress (Q91297702) (← links)
- Nano-delivery of RAD6/Translesion Synthesis Inhibitor SMI#9 for Triple-negative Breast Cancer Therapy (Q91638152) (← links)
- The Mgs1/WRNIP1 ATPase is required to prevent a recombination salvage pathway at damaged replication forks (Q91878890) (← links)
- The nuclear pore complex prevents sister chromatid recombination during replicative senescence (Q92527905) (← links)
- iDamage: a method to integrate modified DNA into the yeast genome (Q92653634) (← links)
- DNA damage tolerance in stem cells, ageing, mutagenesis, disease and cancer therapy (Q93103734) (← links)
- SUMOylation of PCNA by PIAS1 and PIAS4 promotes template switch in the chicken and human B cell lines (Q93379971) (← links)
- An advanced cell cycle tag toolbox reveals principles underlying temporal control of structure-selective nucleases (Q94466519) (← links)