Pages that link to "Q24322944"
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The following pages link to Generation of anti-apoptotic presenilin-2 polypeptides by alternative transcription, proteolysis, and caspase-3 cleavage (Q24322944):
Displaying 50 items.
- Caspase 3 (Q5049057) (← links)
- Presenilin 2 (Q21988969) (← links)
- Cleavage of atrophin-1 at caspase site aspartic acid 109 modulates cytotoxicity (Q22009069) (← links)
- Identification of a novel PSD-95/Dlg/ZO-1 (PDZ)-like protein interacting with the C terminus of presenilin-1 (Q22010752) (← links)
- The novel presenilin-1-associated protein is a proapoptotic mitochondrial protein (Q24310855) (← links)
- Interaction of Alzheimer's presenilin-1 and presenilin-2 with Bcl-X(L). A potential role in modulating the threshold of cell death (Q28142036) (← links)
- Involvement of caspases in proteolytic cleavage of Alzheimer's amyloid-beta precursor protein and amyloidogenic A beta peptide formation (Q28143624) (← links)
- Biochemical and immunocytochemical characterization of calsenilin in mouse brain (Q28220167) (← links)
- Two isoforms of PSAP/MTCH1 share two proapoptotic domains and multiple internal signals for import into the mitochondrial outer membrane (Q28239032) (← links)
- Presenilin 2 deficiency causes a mild pulmonary phenotype and no changes in amyloid precursor protein processing but enhances the embryonic lethal phenotype of presenilin 1 deficiency (Q28505078) (← links)
- Identification of caspases that cleave presenilin-1 and presenilin-2 (Q29999194) (← links)
- Gene expression profile in Alzheimer's brain screened by molecular indexing (Q30168428) (← links)
- The Gtx homeodomain transcription factor exerts neuroprotection using its homeodomain (Q33197281) (← links)
- Alzheimer's disease-associated presenilin 2 interacts with DRAL, an LIM-domain protein (Q33292161) (← links)
- Biology of presenilins as causative molecules for Alzheimer disease. (Q33653958) (← links)
- Molecular genetics of Alzheimer's disease. (Q33842568) (← links)
- Caspases and neurodegeneration: on the cutting edge of new therapeutic approaches. (Q33873247) (← links)
- Presenilin structure, function and role in Alzheimer disease. (Q33971264) (← links)
- ApoptoProteomics, an integrated database for analysis of proteomics data obtained from apoptotic cells (Q34069977) (← links)
- Amyloid precursor protein (APP) and the biology of proteolytic processing: relevance to Alzheimer's disease (Q34208401) (← links)
- Closing in on the amyloid cascade: recent insights into the cell biology of Alzheimer's disease (Q34286933) (← links)
- p53-dependent apoptosis pathways. (Q34305233) (← links)
- Additive regulation of hepatic gene expression by dwarfism and caloric restriction. (Q34546580) (← links)
- Neurons bearing presenilins: weapons for defense or suicide? (Q34687686) (← links)
- Many cuts to ruin: a comprehensive update of caspase substrates (Q35091412) (← links)
- Endometriosis gene expression heterogeneity and biosignature: a phylogenetic analysis (Q35615166) (← links)
- Activation of caspase-3 in single neurons and autophagic granules of granulovacuolar degeneration in Alzheimer's disease. Evidence for apoptotic cell death. (Q35788051) (← links)
- Sox11 Reduces Caspase-6 Cleavage and Activity (Q35822934) (← links)
- Conserved Molecular Underpinnings and Characterization of a Role for Caveolin-1 in the Tumor Microenvironment of Mature T-Cell Lymphomas (Q35840904) (← links)
- Presenilin function and gamma-secretase activity (Q36109130) (← links)
- Apoptotic activities of wild-type and Alzheimer's disease-related mutant presenilins in Drosophila melanogaster (Q36301441) (← links)
- C-terminal maturation fragments of presenilin 1 and 2 control secretion of APP alpha and A beta by human cells and are degraded by proteasome (Q36438184) (← links)
- Caspase-9 mediates synaptic plasticity and memory deficits of Danish dementia knock-in mice: caspase-9 inhibition provides therapeutic protection (Q36526897) (← links)
- The PARL family of mitochondrial rhomboid proteases (Q37666154) (← links)
- Abolishing Tau cleavage by caspases at Aspartate421 causes memory/synaptic plasticity deficits and pre-pathological Tau alterations (Q38634954) (← links)
- Twenty Years of Presenilins--Important Proteins in Health and Disease (Q38938820) (← links)
- γ-Secretase complexes containing caspase-cleaved presenilin-1 increase intracellular Aβ(42) /Aβ(40) ratio (Q39545955) (← links)
- Interference with splicing of Presenilin transcripts has potent dominant negative effects on Presenilin activity (Q40056954) (← links)
- Phosphorylation of presenilin 1 at the caspase recognition site regulates its proteolytic processing and the progression of apoptosis (Q40623499) (← links)
- Apoptosis induced by Na+/H+ antiport inhibition activates the LEI/L-DNase II pathway (Q40650529) (← links)
- Overexpression of wild-type presenilin 2 or its familial Alzheimer's disease-associated mutant does not induce or increase susceptibility to apoptosis in different cell lines. (Q40668193) (← links)
- A loss of function mutant of the presenilin homologue SEL-12 undergoes aberrant endoproteolysis in Caenorhabditis elegans and increases abeta 42 generation in human cells (Q40851871) (← links)
- "Tell me where is calcium bred": clarifying the roles of nuclear calcium (Q41715809) (← links)
- The C-terminal fragment of presenilin 2 triggers p53-mediated staurosporine-induced apoptosis, a function independent of the presenilinase-derived N-terminal counterpart (Q44295644) (← links)
- Characterization of Drosophila Presenilin and its colocalization with Notch during development (Q47959771) (← links)
- Two novel spliced presenilin 2 transcripts in human lymphocyte with oxidant stress and brain (Q48162931) (← links)
- A unifying hypothesis of Alzheimer's disease. III. Risk factors (Q48458709) (← links)
- The presenilin mystery. The research winner-by-a knockout? (Q52576091) (← links)
- Staurosporine-induced activation of caspase-3 is potentiated by presenilin 1 familial Alzheimer's disease mutations in human neuroglioma cells. (Q53231709) (← links)
- The nonconserved hydrophilic loop domain of presenilin (PS) is not required for PS endoproteolysis or enhanced abeta 42 production mediated by familial early onset Alzheimer's disease-linked PS variants. (Q53234112) (← links)