Pages that link to "Q37432581"
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The following pages link to Genome-wide association studies of autoimmune vitiligo identify 23 new risk loci and highlight key pathways and regulatory variants (Q37432581):
Displaying 20 items.
- Genetics of Vitiligo (Q30235209) (← links)
- A case-control study on association of proteasome subunit beta 8 (PSMB8) and transporter associated with antigen processing 1 (TAP1) polymorphisms and their transcript levels in vitiligo from Gujarat (Q33897820) (← links)
- Vitiligo blood transcriptomics provides new insights into disease mechanisms and identifies potential novel therapeutic targets (Q37611016) (← links)
- GWAS summary-based pathway analysis correcting for the genetic confounding impact of environmental exposures. (Q38882795) (← links)
- Vitiligo: Focus on Clinical Aspects, Immunopathogenesis, and Therapy (Q39419931) (← links)
- Improving power of association tests using multiple sets of imputed genotypes from distributed reference panels. (Q40061670) (← links)
- Meeting report: Vitiligo Global Issues Consensus Conference Workshop "Outcome measurement instruments" and Vitiligo International Symposium, Rome, Nov 30-Dec 3rd. (Q40256899) (← links)
- Energetic mitochondrial failing in vitiligo and possible rescue by cardiolipin (Q45070729) (← links)
- Cis-SNPs Set Testing and PrediXcan Analysis for Gene Expression Data using Linear Mixed Models. (Q47142753) (← links)
- Network pharmacological mechanisms of Vernonia anthelmintica (L.) in the treatment of vitiligo: Isorhamnetin induction of melanogenesis via up-regulation of melanin-biosynthetic genes (Q47395062) (← links)
- Expression of inflammasome proteins and inflammasome activation occurs in human, but not in murine keratinocytes (Q47551652) (← links)
- Identification of non-HLA genes associated with development of islet autoimmunity and type 1 diabetes in the prospective TEDDY cohort (Q48522341) (← links)
- More evidence for catalase as a pathogenetic cornerstone in vitiligo (Q50125507) (← links)
- Research Techniques Made Simple: Using Genome-Wide Association Studies to Understand Complex Cutaneous Disorders. (Q52373466) (← links)
- Analysis of predicted loss-of-function variants in UK Biobank identifies variants protective for disease. (Q52565485) (← links)
- Genetic polymorphisms of GZMB and vitiligo: A genetic association study based on Chinese Han population (Q58695433) (← links)
- Lymphoid Stress Surveillance Response Contributes to Vitiligo Pathogenesis (Q59791539) (← links)
- POGLUT1, the putative effector gene driven by rs2293370 in primary biliary cholangitis susceptibility locus chromosome 3q13.33 (Q60909951) (← links)
- Early-onset autoimmune vitiligo associated with an enhancer variant haplotype that upregulates class II HLA expression (Q61124688) (← links)
- Innate lymphocyte-induced CXCR3B-mediated melanocyte apoptosis is a potential initiator of T-cell autoreactivity in vitiligo (Q64053800) (← links)