Ginsenoside Re protects methamphetamine-induced mitochondrial burdens and proapoptosis via genetic inhibition of protein kinase C δ in human neuroblastoma dopaminergic SH-SY5Y cell lines (Q38927155)

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scientific article published on 18 December 2014

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English	Ginsenoside Re protects methamphetamine-induced mitochondrial burdens and proapoptosis via genetic inhibition of protein kinase C δ in human neuroblastoma dopaminergic SH-SY5Y cell lines	scientific article published on 18 December 2014

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scholarly article

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Europe PubMed Central

PubMed publication ID

7 September 2017

Ginsenoside Re protects methamphetamine-induced mitochondrial burdens and proapoptosis via genetic inhibition of protein kinase C δ in human neuroblastoma dopaminergic SH-SY5Y cell lines (English)

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Europe PubMed Central

PubMed publication ID

7 September 2017

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author name string

Yunsung Nam

1

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7 September 2017

Myung Bok Wie

2

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7 September 2017

Eun-Joo Shin

3

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7 September 2017

Thuy-Ty Lan Nguyen

4

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7 September 2017

Seung-Yeol Nah

5

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7 September 2017

Sung Kwon Ko

6

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7 September 2017

Ji Hoon Jeong

7

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7 September 2017

Choon-Gon Jang

8

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7 September 2017

Hyoung-Chun Kim

9

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7 September 2017

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publication date

18 December 2014

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7 September 2017

Journal of Applied Toxicology

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7 September 2017

35

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7 September 2017

8

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7 September 2017

927-944

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PubMed publication ID

7 September 2017

Proteomic analysis of dopamine and α-synuclein interplay in a cellular model of Parkinson's disease pathogenesis

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Dopamine- or L-DOPA-induced neurotoxicity: the role of dopamine quinone formation and tyrosinase in a model of Parkinson's disease

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Electrophysiological effects of ginseng and ginsenoside Re in guinea pig ventricular myocytes

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Nitric oxide-dependent modulation of the delayed rectifier K+ current and the L-type Ca2+ current by ginsenoside Re, an ingredient of Panax ginseng, in guinea-pig cardiomyocytes

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Two Faces of Protein Kinase Cδ: The Contrasting Roles of PKCδ in Cell Survival and Cell Death

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Involvement of dopamine receptors in binge methamphetamine-induced activation of endoplasmic reticulum and mitochondrial stress pathways

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Protein kinase C delta is required for p47phox phosphorylation and translocation in activated human monocytes

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Methamphetamine-induced neuronal apoptosis involves the activation of multiple death pathways. Review

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Novel imine antioxidants at low nanomolar concentrations protect dopaminergic cells from oxidative neurotoxicity

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Methamphetamine induces heme oxygenase-1 expression in cortical neurons and glia to prevent its toxicity.

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Human DJ-1-specific Transcriptional Activation of Tyrosine Hydroxylase Gene

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Methamphetamine induces neuronal apoptosis via cross-talks between endoplasmic reticulum and mitochondria-dependent death cascades

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Pharmacokinetic study of ginsenoside Re with pure ginsenoside Re and ginseng berry extracts in mouse using ultra performance liquid chromatography/mass spectrometric method

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Methamphetamine induces autophagy and apoptosis in a mesencephalic dopaminergic neuronal culture model: role of cathepsin-D in methamphetamine-induced apoptotic cell death

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Neuroprotective effect of resveratrol against methamphetamine-induced dopaminergic apoptotic cell death in a cell culture model of neurotoxicity.

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Role of Proteolytic Activation of Protein Kinase Cδ in Oxidative Stress-Induced Apoptosis

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Caspase‐3 dependent proteolytic activation of protein kinase Cδ mediates and regulates 1‐methyl‐4‐phenylpyridinium (MPP+)‐induced apoptotic cell death in dopaminergic cells: relevance to oxidative stress in dopaminergic degeneration

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A Specific Light Chain of Kinesin Associates with Mitochondria in Cultured Cells

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Pharmacological action of Panax ginseng on the behavioral toxicities induced by psychotropic agents

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Effects of ginsenosides on Ca2+ channels and membrane capacitance in rat adrenal chromaffin cells.

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Rescue of PINK1 protein null-specific mitochondrial complex IV deficits by ginsenoside Re activation of nitric oxide signaling

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Methamphetamine-mediated modulation of MOR expression in the SH-SY5Y neuroblastoma cell line

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Involvement of protein kinase C delta in the alteration of mitochondrial mass in human cells under oxidative stress.

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Evaluation of the neurotoxic/neuroprotective role of organoselenides using differentiated human neuroblastoma SH-SY5Y cell line challenged with 6-hydroxydopamine

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Ginseng compounds: an update on their molecular mechanisms and medical applications

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Mitochondrial glutathione: features, regulation and role in disease

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Brain-derived neurotrophic factor rescues neuronal death induced by methamphetamine

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Parkinson's disease: don't mess with calcium

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Evidence for synaptic apoptosis

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Methamphetamine blood concentrations in human abusers: application to pharmacokinetic modeling

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Methamphetamine induces macropinocytosis in differentiated SH-SY5Y human neuroblastoma cells

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Mitochondrial calcium function and dysfunction in the central nervous system

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Studies and perspectives of protein kinase C.

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Intracellular signaling by hydrolysis of phospholipids and activation of protein kinase C

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Escalating dose pretreatment induces pharmacodynamic and not pharmacokinetic tolerance to a subsequent high-dose methamphetamine binge.

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Inhibition of inflammations and macrophage activation by ginsenoside-Re isolated from Korean ginseng (Panax ginseng C.A. Meyer).

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21 January 2018

Melatonin attenuates methamphetamine-induced disturbances in mitochondrial dynamics and degeneration in neuroblastoma SH-SY5Y cells

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The anti-inflammatory effect of melatonin on methamphetamine-induced proinflammatory mediators in human neuroblastoma dopamine SH-SY5Y cell lines

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Mortalin overexpression attenuates beta-amyloid-induced neurotoxicity in SH-SY5Y cells.

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PKCδ inhibition enhances tyrosine hydroxylase phosphorylation in mice after methamphetamine treatment.

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Role of oxidative stress in methamphetamine-induced dopaminergic toxicity mediated by protein kinase Cδ.

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Kainate-induced mitochondrial oxidative stress contributes to hippocampal degeneration in senescence-accelerated mice

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Protection against kainate neurotoxicity by ginsenosides: attenuation of convulsive behavior, mitochondrial dysfunction, and oxidative stress

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Ginsenosides attenuate methamphetamine-induced behavioral side effects in mice via activation of adenosine A2A receptors: possible involvements of the striatal reduction in AP-1 DNA binding activity and proenkephalin gene expression

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Ginsenoside Re rescues methamphetamine-induced oxidative damage, mitochondrial dysfunction, microglial activation, and dopaminergic degeneration by inhibiting the protein kinase Cδ gene

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Protective potential of IL-6 against trimethyltin-induced neurotoxicity in vivo.

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Protein kinase C-epsilon protects PC12 cells against methamphetamine-induced death: possible involvement of suppression of glutamate receptor

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Involvement of oxidative stress-activated JNK signaling in the methamphetamine-induced cell death of human SH-SY5Y cells.

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Melatonin reduces induction of Bax, caspase and cell death in methamphetamine-treated human neuroblastoma SH-SY5Y cultured cells

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Protective effects of pseudoginsenoside-F11 on methamphetamine-induced neurotoxicity in mice

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Enhanced oxidative stress and aberrant mitochondrial biogenesis in human neuroblastoma SH-SY5Y cells during methamphetamine induced apoptosis.

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Antioxidant effects of ginsenoside Re in cardiomyocytes.

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Effects of manganese on tyrosine hydroxylase (TH) activity and TH-phosphorylation in a dopaminergic neural cell line

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Methamphetamine-induced cell death: selective vulnerability in neuronal subpopulations of the striatum in mice

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Methamphetamine-induced striatal apoptosis in the mouse brain: comparison of a binge to an acute bolus drug administration

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Quantitative Analysis of Ginsenosides Composition in Flower Buds of Various Ginseng Plants

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p73β Is Regulated by Protein Kinase Cδ Catalytic Fragment Generated in the Apoptotic Response to DNA Damage

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Identifiers

10.1002/JAT.3093

1 reference

Europe PubMed Central

PubMed publication ID

7 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

7 September 2017

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