The long physiological reach of the yeast vacuolar H+-ATPase (Q37000446)

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scientific article published on December 2007

Language	Label	Description	Also known as
English	The long physiological reach of the yeast vacuolar H+-ATPase	scientific article published on December 2007

Statements

scholarly article

1 reference

Europe PubMed Central

PMC publication ID

17 August 2017

1 reference

Europe PubMed Central

The long physiological reach of the yeast vacuolar H+-ATPase (English)

1 reference

Europe PubMed Central

PMC publication ID

17 August 2017

author name string

Patricia M Kane

1

1 reference

Europe PubMed Central

PMC publication ID

17 August 2017

publication date

1 December 2007

1 reference

Europe PubMed Central

PMC publication ID

17 August 2017

Journal of Bioenergetics and Biomembranes

1 reference

Europe PubMed Central

PMC publication ID

17 August 2017

39

1 reference

Europe PubMed Central

PMC publication ID

17 August 2017

5-6

1 reference

Europe PubMed Central

PMC publication ID

17 August 2017

415-421

1 reference

Europe PubMed Central

PMC publication ID

17 August 2017

A phenomics approach in yeast links proton and calcium pump function in the Golgi

1 reference

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Genomic screening in vivo reveals the role played by vacuolar H+ ATPase and cytosolic acidification in sensitivity to DNA-damaging agents such as cisplatin

1 reference

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Glutaredoxins Grx3 and Grx4 regulate nuclear localisation of Aft1 and the oxidative stress response in Saccharomyces cerevisiae.

1 reference

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New insights into the regulation of V-ATPase-dependent proton secretion

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Free radicals and antioxidants in normal physiological functions and human disease

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PKR1 encodes an assembly factor for the yeast V-type ATPase.

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Yeast genes involved in response to lactic acid and acetic acid: acidic conditions caused by the organic acids in Saccharomyces cerevisiae cultures induce expression of intracellular metal metabolism genes regulated by Aft1p

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The where, when, and how of organelle acidification by the yeast vacuolar H+-ATPase

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Complex cellular responses to reactive oxygen species

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A genomic screen for yeast vacuolar membrane ATPase mutants

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Cytosolic thioredoxin peroxidase I and II are important defenses of yeast against organic hydroperoxide insult: catalases and peroxiredoxins cooperate in the decomposition of H2O2 by yeast

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Cells have distinct mechanisms to maintain protection against different reactive oxygen species: oxidative-stress-response genes

1 reference

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Copper and iron are the limiting factors for growth of the yeast Saccharomyces cerevisiae in an alkaline environment.

1 reference

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The yeast vacuolar proton-translocating ATPase contains a subunit homologous to the Manduca sexta and bovine e subunits that is essential for function

1 reference

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Metal-responsive transcription factors that regulate iron, zinc, and copper homeostasis in eukaryotic cells.

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Integration of chemical-genetic and genetic interaction data links bioactive compounds to cellular target pathways

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Pathways of Oxidative Damage

1 reference

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Inherited disorders of the H+-ATPase

1 reference

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Functional profiling of the Saccharomyces cerevisiae genome

1 reference

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Genomic screen for vacuolar protein sorting genes in Saccharomyces cerevisiae

1 reference

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1 reference

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Genomic analysis of homotypic vacuole fusion

1 reference

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The vacuolar (H+)-ATPases--nature's most versatile proton pumps

1 reference

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Animal plasma membrane energization by proton-motive V-ATPases

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Defects in the yeast high affinity iron transport system result in increased metal sensitivity because of the increased expression of transporters with a broad transition metal specificity

1 reference

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Assembly of the yeast vacuolar H+-ATPase occurs in the endoplasmic reticulum and requires a Vma12p/Vma22p assembly complex

1 reference

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Oxidative stress: the paradox of aerobic life

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1 reference

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1 reference

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1 reference

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1 reference

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Inhibitors of vacuolar-type H(+)-ATPase suppresses proliferation of cultured cells

1 reference

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Saccharomyces cerevisiae glutaredoxin 5-deficient cells subjected to continuous oxidizing conditions are affected in the expression of specific sets of genes

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Yap1 and Skn7 control two specialized oxidative stress response regulons in yeast.

1 reference

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4 September 2018

Loss of vacuolar proton-translocating ATPase activity in yeast results in chronic oxidative stress

1 reference

https://pubmed.ncbi.nlm.nih.gov/18000744

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Trans-complex formation by proteolipid channels in the terminal phase of membrane fusion

1 reference

https://pubmed.ncbi.nlm.nih.gov/18000744

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Vacuolar H(+)-ATPase, but not mitochondrial F(1)F(0)-ATPase, is required for NaCl tolerance in Saccharomyces cerevisiae

1 reference

https://pubmed.ncbi.nlm.nih.gov/18000744

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Degradation of misfolded proteins prevents ER-derived oxidative stress and cell death

1 reference

https://pubmed.ncbi.nlm.nih.gov/18000744

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

The vacuolar H(+)-ATPase of Saccharomyces cerevisiae is required for efficient copper detoxification, mitochondrial function, and iron metabolism

1 reference

https://pubmed.ncbi.nlm.nih.gov/18000744

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Relative roles of Na+/H+ exchange and vacuolar-type H+ ATPases in regulating cytoplasmic pH and function in murine peritoneal macrophages

1 reference

https://pubmed.ncbi.nlm.nih.gov/18000744

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Specific inhibitors of vacuolar H(+)-ATPase trigger apoptotic cell death of osteoclasts

1 reference

https://pubmed.ncbi.nlm.nih.gov/18000744

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1007/S10863-007-9112-Z

1 reference

Europe PubMed Central

PMC publication ID

17 August 2017

PMC publication ID

1 reference

Europe PubMed Central

PMC publication ID

17 August 2017

PubMed publication ID

1 reference

Europe PubMed Central

PMC publication ID

17 August 2017

ResearchGate publication ID

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