Partial inhibition of the ubiquitin-proteasome system ameliorates cardiac dysfunction following ischemia-reperfusion in the presence of high glucose (Q35896759)

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scientific article published on 28 July 2015

Language	Label	Description	Also known as
English	Partial inhibition of the ubiquitin-proteasome system ameliorates cardiac dysfunction following ischemia-reperfusion in the presence of high glucose	scientific article published on 28 July 2015

Statements

scholarly article

1 reference

Europe PubMed Central

PMC publication ID

11 August 2017

Partial inhibition of the ubiquitin-proteasome system ameliorates cardiac dysfunction following ischemia-reperfusion in the presence of high glucose (English)

1 reference

Europe PubMed Central

PMC publication ID

11 August 2017

ubiquitin-proteasome system

0 references

author name string

Buin Adams

1

1 reference

Europe PubMed Central

PMC publication ID

11 August 2017

Rudo F Mapanga

2

1 reference

Europe PubMed Central

PMC publication ID

11 August 2017

M Faadiel Essop

3

1 reference

Europe PubMed Central

PMC publication ID

11 August 2017

language of work or name

0 references

publication date

28 July 2015

1 reference

Europe PubMed Central

PMC publication ID

11 August 2017

Cardiovascular Diabetology

1 reference

Europe PubMed Central

PMC publication ID

11 August 2017

14

1 reference

Europe PubMed Central

PMC publication ID

11 August 2017

94

1 reference

Europe PubMed Central

PMC publication ID

11 August 2017

https://scigraph.springernature.com/pub.10.1186/s12933-015-0258-4

0 references

The benefits of tight glycemic control in critical illness: Sweeter than assumed?

1 reference

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11 July 2018

Type 2 diabetes and cardiovascular disease: Have all risk factors the same strength?

1 reference

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11 July 2018

Resveratrol-enhanced autophagic flux ameliorates myocardial oxidative stress injury in diabetic mice

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4517635

11 July 2018

Proteasomal and lysosomal protein degradation and heart disease

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4517635

11 July 2018

The ubiquitin proteasome system and myocardial ischemia

1 reference

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11 July 2018

Oleanolic acid: a novel cardioprotective agent that blunts hyperglycemia-induced contractile dysfunction.

1 reference

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11 July 2018

Genetically induced moderate inhibition of the proteasome in cardiomyocytes exacerbates myocardial ischemia-reperfusion injury in mice

1 reference

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11 July 2018

Molecular chaperones in targeting misfolded proteins for ubiquitin-dependent degradation

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Proteasome malfunction activates macroautophagy in the heart

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Photodynamic therapy based on 5‐aminolevulinic acid and its use as an antimicrobial Agent

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11 July 2018

Role of Chronic and Acute Hyperglycemia in the Development of Diabetes Complications

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11 July 2018

Glucose control in hospitalized patients.

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11 July 2018

Hyperglycemia-mediated activation of the hexosamine biosynthetic pathway results in myocardial apoptosis

1 reference

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11 July 2018

Sent to destroy: the ubiquitin proteasome system regulates cell signaling and protein quality control in cardiovascular development and disease

1 reference

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11 July 2018

Mechanisms of cross-talk between the ubiquitin-proteasome and autophagy-lysosome systems

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11 July 2018

Proteasome inhibition during myocardial infarction

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4517635

11 July 2018

Metabolic reserve of the heart: the forgotten link between contraction and coronary flow

1 reference

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20S proteasome and its inhibitors: crystallographic knowledge for drug development

1 reference

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11 July 2018

The ubiquitin-proteasome system in cardiac physiology and pathology

1 reference

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11 July 2018

The ubiquitin-proteasome system: focus on the heart.

1 reference

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11 July 2018

Ubiquitination of Keap1, a BTB-Kelch substrate adaptor protein for Cul3, targets Keap1 for degradation by a proteasome-independent pathway

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4517635

11 July 2018

The pathobiology of diabetic complications: a unifying mechanism

1 reference

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Protection against electrophile and oxidative stress by induction of phase 2 genes: the quest for the elusive sensor that responds to inducers.

1 reference

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11 July 2018

Oxidative stress sensor Keap1 functions as an adaptor for Cul3-based E3 ligase to regulate proteasomal degradation of Nrf2

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4517635

11 July 2018

Unique function of the Nrf2-Keap1 pathway in the inducible expression of antioxidant and detoxifying enzymes

1 reference

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11 July 2018

Keap1-dependent proteasomal degradation of transcription factor Nrf2 contributes to the negative regulation of antioxidant response element-driven gene expression

1 reference

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11 July 2018

Proteasome inhibitors: from research tools to drug candidates

1 reference

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11 July 2018

Stress hyperglycaemia and increased risk of death after myocardial infarction in patients with and without diabetes: a systematic overview

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11 July 2018

Intensive blood-glucose control with sulphonylureas or insulin compared with conventional treatment and risk of complications in patients with type 2 diabetes (UKPDS 33). UK Prospective Diabetes Study (UKPDS) Group

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Lactacystin and clasto-lactacystin beta-lactone modify multiple proteasome beta-subunits and inhibit intracellular protein degradation and major histocompatibility complex class I antigen presentation

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The effect of intensive treatment of diabetes on the development and progression of long-term complications in insulin-dependent diabetes mellitus. The Diabetes Control and Complications Trial Research Group

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Inhibition of proteasome activities and subunit-specific amino-terminal threonine modification by lactacystin

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Role of diabetes in congestive heart failure: the Framingham study

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Detrimental effects of acute hyperglycaemia on the rat heart.

1 reference

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Therapeutic effect of MG-132 on diabetic cardiomyopathy is associated with its suppression of proteasomal activities: roles of Nrf2 and NF-κB.

1 reference

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The hexosamine biosynthetic pathway can mediate myocardial apoptosis in a rat model of diet-induced insulin resistance.

1 reference

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Tight glycemic control reduces heart inflammation and remodeling during acute myocardial infarction in hyperglycemic patients.

1 reference

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The ubiquitin-proteasome system contributes to the inflammatory injury in ischemic diabetic myocardium: the role of glycemic control

1 reference

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Hyperglycemia as a predictor of in-hospital mortality in elderly patients without diabetes mellitus admitted to a sub-intensive care unit

1 reference

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Proteasome inhibition 1 h following ischemia protects GRK2 and prevents malignant ventricular tachyarrhythmias and SCD in a model of myocardial infarction

1 reference

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Proteasome inhibition attenuates infarct size and preserves cardiac function in a murine model of myocardial ischemia-reperfusion injury.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4517635

24 September 2018

Proteasome mediates removal of proteins oxidized during myocardial ischemia

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4517635

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Oxidized and ubiquitinated proteins may predict recovery of postischemic cardiac function: essential role of the proteasome.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4517635

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Protein kinase Cdelta activation induces apoptosis in response to cardiac ischemia and reperfusion damage: a mechanism involving BAD and the mitochondria

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4517635

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Proteasome inhibition ablates activation of NF-kappa B in myocardial reperfusion and reduces reperfusion injury

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4517635

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Glucose metabolism in patients with acute myocardial infarction and no previous diagnosis of diabetes mellitus: a prospective study

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4517635

24 September 2018

Inhibition of the ubiquitin-proteasome pathway induces differential heat-shock protein response in cardiomyocytes and renders early cardiac protection

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4517635

24 September 2018

Oxidative modification and inactivation of the proteasome during coronary occlusion/reperfusion

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4517635

24 September 2018

Identifiers

10.1186/S12933-015-0258-4

1 reference

Europe PubMed Central

PMC publication ID

11 August 2017

PMC publication ID

1 reference

Europe PubMed Central

PMC publication ID

11 August 2017

PubMed publication ID

1 reference

Europe PubMed Central

PMC publication ID

11 August 2017

ResearchGate publication ID

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