Mitochondrial intermembrane junctional complexes and their involvement in cell death (Q34648830)

3 August 2017

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title

Mitochondrial intermembrane junctional complexes and their involvement in cell death (English)

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3 August 2017

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Martin Crompton

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3 August 2017

Emma Barksby

series ordinal

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3 August 2017

Nicholas Johnson

series ordinal

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3 August 2017

Michela Capano

series ordinal

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3 August 2017

publication date

1 February 2002

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3 August 2017

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3 August 2017

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3 August 2017

issue

2-3

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3 August 2017

page(s)

143-152

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Chemical and physical fixation of isolated mitochondria in low-energy and high-energy states

3 August 2017

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Subcellular distribution of rat liver porin

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The role of creatine kinase in inhibition of mitochondrial permeability transition

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Immunoelectron microscopic study of the distribution of porin on outer membranes of rat heart mitochondria

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The mitochondrial permeability transition pore and its role in cell death

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Mitochondrial peripheral-type benzodiazepine receptor expression. Correlation with gonadotropin-releasing hormone (GnRH) agonist-induced apoptosis in the corpus luteum

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Tissue-specific transcription pattern of the adenine nucleotide translocase isoforms in humans.

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A novel isoform of the mitochondrial outer membrane protein VDAC3 via alternative splicing of a 3-base exon. Functional characteristics and subcellular localization

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Import and processing of heart mitochondrial cyclophilin D

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Cyclophilin-D binds strongly to complexes of the voltage-dependent anion channel and the adenine nucleotide translocase to form the permeability transition pore

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Cyclophilin catalyzes protein folding in yeast mitochondria

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Human T cell cyclophilin18 binds to thiol-specific antioxidant protein Aop1 and stimulates its activity

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Evidence for the Involvement of a Membrane-Associated Cyclosporin-A-Binding Protein in the Ca2+-Activated Inner Membrane Pore of Heart Mitochondria

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Complexes between porin, hexokinase, mitochondrial creatine kinase and adenylate translocator display properties of the permeability transition pore. Implication for regulation of permeability transition by the kinases

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On the involvement of a cyclosporin A sensitive mitochondrial pore in myocardial reperfusion injury

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7 January 2021

The pro-apoptotic proteins, Bid and Bax, cause a limited permeabilization of the mitochondrial outer membrane that is enhanced by cytosol

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7 January 2021

Conformation of the Bax C-terminus regulates subcellular location and cell death

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7 January 2021

Bax is present as a high molecular weight oligomer/complex in the mitochondrial membrane of apoptotic cells

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7 January 2021

Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC

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7 January 2021

Bax and adenine nucleotide translocator cooperate in the mitochondrial control of apoptosis

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7 January 2021

Adenine nucleotide translocase-1, a component of the permeability transition pore, can dominantly induce apoptosis

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7 January 2021

Hepatitis B virus X protein colocalizes to mitochondria with a human voltage-dependent anion channel, HVDAC3, and alters its transmembrane potential

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7 January 2021

Neisseria meningitidis porin PorB interacts with mitochondria and protects cells from apoptosis

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7 January 2021

Regulated targeting of BAX to mitochondria.

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7 January 2021

Enforced dimerization of BAX results in its translocation, mitochondrial dysfunction and apoptosis

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7 January 2021

Functional consequences of the sustained or transient activation by Bax of the mitochondrial permeability transition pore

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7 January 2021

Bax-induced cytochrome C release from mitochondria is independent of the permeability transition pore but highly dependent on Mg2+ ions

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7 January 2021

Kinetic evidence for a heart mitochondrial pore activated by Ca2+, inorganic phosphate and oxidative stress. A potential mechanism for mitochondrial dysfunction during cellular Ca2+ overload

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7 January 2021

Correlation between cytosolic free calcium, contracture, ATP, and irreversible ischemic injury in perfused rat heart

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7 January 2021

Cytosolic free Ca2+ in single rat heart cells during anoxia and reoxygenation

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7 January 2021

Mitochondrial non-specific pores remain closed during cardiac ischaemia, but open upon reperfusion

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7 January 2021

Mitochondrial intermembrane junctional complexes and their role in cell death

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7 January 2021

Transient mitochondrial depolarizations reflect focal sarcoplasmic reticular calcium release in single rat cardiomyocytes

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7 January 2021

Cytochrome c release occurs via Ca2+-dependent and Ca2+-independent mechanisms that are regulated by Bax.

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7 January 2021

10.1016/S0300-9084(02)01368-8

Identifiers

DOI

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3 August 2017

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