Lamotrigine reduces spontaneous and evoked GABAA receptor-mediated synaptic transmission in the basolateral amygdala: implications for its effects in seizure and affective disorders (Q34123860): Difference between revisions

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scientific article

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English	Lamotrigine reduces spontaneous and evoked GABAA receptor-mediated synaptic transmission in the basolateral amygdala: implications for its effects in seizure and affective disorders	scientific article

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

31 July 2017

Lamotrigine reduces spontaneous and evoked GABAA receptor-mediated synaptic transmission in the basolateral amygdala: implications for its effects in seizure and affective disorders (English)

1 reference

Europe PubMed Central

PubMed publication ID

31 July 2017

1 reference

based on heuristic

inferred from title

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based on heuristic

inferred from title

author name string

M F M Braga

1

1 reference

Europe PubMed Central

PubMed publication ID

31 July 2017

V Aroniadou-Anderjaska

2

1 reference

Europe PubMed Central

PubMed publication ID

31 July 2017

R M Post

3

1 reference

Europe PubMed Central

PubMed publication ID

31 July 2017

H Li

4

1 reference

Europe PubMed Central

PubMed publication ID

31 July 2017

publication date

1 March 2002

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Europe PubMed Central

PubMed publication ID

31 July 2017

Neuropharmacology

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Europe PubMed Central

PubMed publication ID

31 July 2017

42

1 reference

Europe PubMed Central

PubMed publication ID

31 July 2017

4

1 reference

Europe PubMed Central

PubMed publication ID

31 July 2017

522-529

1 reference

Europe PubMed Central

PubMed publication ID

31 July 2017

Input-specific LTP and depotentiation in the basolateral amygdala

1 reference

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Unitary synaptic currents between lacunosum-moleculare interneurones and pyramidal cells in rat hippocampus

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A Double-Blind, Placebo-Controlled, Prophylaxis Study of Lamotrigine in Rapid-Cycling Bipolar Disorder

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An in vitro electrophysiological study on the effects of phenytoin, lamotrigine and gabapentin on striatal neurons

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An in vitro investigation of the action of lamotrigine on neuronal voltage-activated sodium channels

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The anticonvulsant, lamotrigine decreases spontaneous glutamate release but increases spontaneous GABA release in the rat entorhinal cortex in vitro

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Paired-pulse facilitation and depression at unitary synapses in rat hippocampus: quantal fluctuation affects subsequent release.

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Clozapine plus lamotrigine in treatment-resistant schizophrenia.

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Lamotrigine for the treatment of bipolar disorder

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Lamotrigine in the treatment of schizoaffective disorder

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Lamotrigine treatment of refractory bipolar disorder

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Lamotrigine. A review of its pharmacological properties and clinical efficacy in epilepsy

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Paired-pulse modulation at individual GABAergic synapses in rat hippocampus

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An open study of lamotrigine in refractory bipolar depression.

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Pharmacological studies on lamotrigine, a novel potential antiepileptic drug: II. Neurochemical studies on the mechanism of action

1 reference

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Studies on the mechanism of action of the novel anticonvulsant lamotrigine (Lamictal) using primary neuroglial cultures from rat cortex

1 reference

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Modulation of synaptic transmission and long-term potentiation: effects on paired pulse facilitation and EPSC variance in the CA1 region of the hippocampus

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Lamotrigine in absence and primary generalized epilepsies

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GABA and mood disorders: a brief review and hypothesis

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Lamotrigine treatment during amygdala-kindled seizure development fails to inhibit seizures and diminishes subsequent anticonvulsant efficacy

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Synaptic plasticity in the basolateral amygdala in transgenic mice expressing dominant-negative cAMP response element-binding protein (CREB) in forebrain.

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The anticonvulsant lamotrigine in treatment-resistant manic-depressive illness.

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A review of evidence for GABergic predominance/glutamatergic deficit as a common etiological factor in both schizophrenia and affective psychoses: More support for a continuum hypothesis of ?functional? psychosis

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Differential inhibition by riluzole, lamotrigine, and phenytoin of sodium and calcium currents in cortical neurons: implications for neuroprotective strategies

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Lamotrigine for generalised epilepsies

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Effect of lamotrigine on the electrically-evoked release of endogenous amino acids from slices of dorsal horn of the rat spinal cord

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Large, deep layer pyramid-pyramid single axon EPSPs in slices of rat motor cortex display paired pulse and frequency-dependent depression, mediated presynaptically and self-facilitation, mediated postsynaptically

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Lamotrigine in primary generalised epilepsy

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Differential Treatment of Bipolar Disorder with Old and New Antiepileptic Drugs

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Similar potency of carbamazepine, oxcarbazepine, and lamotrigine in inhibiting the release of glutamate and other neurotransmitters

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Inhibition of N-type calcium currents by lamotrigine in rat amygdalar neurones

1 reference

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Presynaptic inhibition of excitatory neurotransmission by lamotrigine in the rat amygdalar neurons

1 reference

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Kindling: separate vs. shared mechanisms in affective disorders and epilepsy

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Cellular and molecular actions of lamotrigine: Possible mechanisms of efficacy in bipolar disorder

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Interaction of the antiepileptic drug lamotrigine with recombinant rat brain type IIA Na+ channels and with native Na+ channels in rat hippocampal neurones

1 reference

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7 January 2021

Identifiers

10.1016/S0028-3908(01)00198-8

1 reference

Europe PubMed Central

PubMed publication ID

31 July 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

31 July 2017

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