APLF (C2orf13) is a novel human protein involved in the cellular response to chromosomal DNA strand breaks (Q24299831)

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Language	Label	Description	Also known as
English	APLF (C2orf13) is a novel human protein involved in the cellular response to chromosomal DNA strand breaks	scientific article

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scholarly article

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APLF (C2orf13) is a novel human protein involved in the cellular response to chromosomal DNA strand breaks (English)

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author name string

Natasha Iles

1

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Stuart Rulten

2

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Sherif F El-Khamisy

3

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Keith W Caldecott

4

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language of work or name

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publication date

May 2007

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Molecular and Cellular Biology

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27

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10

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3793-803

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The neurodegenerative disease protein aprataxin resolves abortive DNA ligation intermediates

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The FHA domain of aprataxin interacts with the C-terminal region of XRCC1

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Xrcc4 physically links DNA end processing by polynucleotide kinase to DNA ligation by DNA ligase IV

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The ataxia-oculomotor apraxia 1 gene product has a role distinct from ATM and interacts with the DNA strand break repair proteins XRCC1 and XRCC4

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Tristetraprolin and Its Family Members Can Promote the Cell-Free Deadenylation of AU-Rich Element-Containing mRNAs by Poly(A) Ribonuclease

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Involvement of human polynucleotide kinase in double-strand break repair by non-homologous end joining

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A cell cycle-specific requirement for the XRCC1 BRCT II domain during mammalian DNA strand break repair

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Megabase chromatin domains involved in DNA double-strand breaks in vivo

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Molecular cloning of the human XRCC1 gene, which corrects defective DNA strand break repair and sister chromatid exchange

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DNA double-stranded breaks induce histone H2AX phosphorylation on serine 139

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A monomeric red fluorescent protein

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The protein kinase CK2 facilitates repair of chromosomal DNA single-strand breaks

1 reference

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Tristetraprolin and other CCCH tandem zinc-finger proteins in the regulation of mRNA turnover

2 references

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19 March 2017

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The gene mutated in ataxia-ocular apraxia 1 encodes the new HIT/Zn-finger protein aprataxin

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Early-onset ataxia with ocular motor apraxia and hypoalbuminemia is caused by mutations in a new HIT superfamily gene

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XRCC1 stimulates human polynucleotide kinase activity at damaged DNA termini and accelerates DNA single-strand break repair

1 reference

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The XRCC4 gene encodes a novel protein involved in DNA double-strand break repair and V(D)J recombination

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Structural basis for phosphorylation-dependent signaling in the DNA-damage response

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The molecular architecture of the mammalian DNA repair enzyme, polynucleotide kinase

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A new XRCC1-containing complex and its role in cellular survival of methyl methanesulfonate treatment

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In situ analysis of repair processes for oxidative DNA damage in mammalian cells.

1 reference

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XRCC1 and DNA strand break repair

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DNA single-strand break repair and spinocerebellar ataxia

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Mutation of a BRCT domain selectively disrupts DNA single-strand break repair in noncycling Chinese hamster ovary cells

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XRCC1 keeps DNA from getting stranded

1 reference

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Identification of proteins that interact with CREB during differentiation of F9 embryonal carcinoma cells

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X-ray induced DNA double strand break production and repair in mammalian cells as measured by neutral filter elution

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Construction of a functional cDNA clone of the hamster ERCC2 DNA repair and transcription gene

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A CHO-cell strain having hypersensitivity to mutagens, a defect in DNA strand-break repair, and an extraordinary baseline frequency of sister-chromatid exchange

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Isolation of cell cycle-dependent gamma ray-sensitive Chinese hamster ovary cell

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Measurement of chromosomal DNA single-strand breaks and replication fork progression rates.

1 reference

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29 November 2018

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