Critical role of Ena/VASP proteins for filopodia formation in neurons and in function downstream of netrin-1. (Q44831766)

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scientific article published in April 2004

Language	Label	Description	Also known as
English	Critical role of Ena/VASP proteins for filopodia formation in neurons and in function downstream of netrin-1.	scientific article published in April 2004

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

6 December 2017

Critical role of Ena/VASP proteins for filopodia formation in neurons and in function downstream of netrin-1. (English)

1 reference

Europe PubMed Central

PubMed publication ID

6 December 2017

7

1 reference

Europe PubMed Central

PubMed publication ID

6 December 2017

Matthias Krause

object named as

Matthias Krause

5

0 references

object named as

Erik W Dent

2

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object named as

Cecile Lebrand

1

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author name string

Geraldine A Strasser

3

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Europe PubMed Central

PubMed publication ID

6 December 2017

Lorene M Lanier

4

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Europe PubMed Central

PubMed publication ID

6 December 2017

Tatyana M Svitkina

6

1 reference

Europe PubMed Central

PubMed publication ID

6 December 2017

Frank B Gertler

8

1 reference

Europe PubMed Central

PubMed publication ID

6 December 2017

language of work or name

0 references

publication date

1 April 2004

1 reference

Europe PubMed Central

PubMed publication ID

6 December 2017

1 reference

Europe PubMed Central

PubMed publication ID

6 December 2017

42

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Europe PubMed Central

PubMed publication ID

6 December 2017

1

1 reference

Europe PubMed Central

PubMed publication ID

6 December 2017

37-49

1 reference

Europe PubMed Central

PubMed publication ID

6 December 2017

The vasodilator-stimulated phosphoprotein (VASP) is involved in cGMP- and cAMP-mediated inhibition of agonist-induced platelet aggregation, but is dispensable for smooth muscle function.

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Negative regulation of fibroblast motility by Ena/VASP proteins

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Regulating cellular actin assembly

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Antagonism between Ena/VASP proteins and actin filament capping regulates fibroblast motility

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Disoriented pathfinding by pioneer neurone growth cones deprived of filopodia by cytochalasin treatment

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Turning of retinal growth cones in a netrin-1 gradient mediated by the netrin receptor DCC.

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Molecular mechanisms of axon guidance

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Netrin-1-mediated axon outgrowth requires deleted in colorectal cancer-dependent MAPK activation

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Actions of cytochalasins on the organization of actin filaments and microtubules in a neuronal growth cone

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enabled, a dosage-sensitive suppressor of mutations in the Drosophila Abl tyrosine kinase, encodes an Abl substrate with SH3 domain-binding properties.

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Mena, a relative of VASP and Drosophila Enabled, is implicated in the control of microfilament dynamics

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The netrin receptor UNC-40/DCC stimulates axon attraction and outgrowth through enabled and, in parallel, Rac and UNC-115/AbLIM.

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Ena/VASP proteins regulate cortical neuronal positioning

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Experimental observations on the development of polarity by hippocampal neurons in culture

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Requirement of a vasodilator-stimulated phosphoprotein family member for cell adhesion, the formation of filopodia, and chemotaxis in dictyostelium

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Identifiers

10.1016/S0896-6273(04)00108-4

1 reference

Europe PubMed Central

PubMed publication ID

6 December 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

6 December 2017

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