Enhanced glycogen synthase kinase-3beta activity mediates hypoxia-induced apoptosis of vascular smooth muscle cells and is prevented by glucose transport and metabolism (Q44117700)

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scientific article published on 27 August 2002

Language	Label	Description	Also known as
English	Enhanced glycogen synthase kinase-3beta activity mediates hypoxia-induced apoptosis of vascular smooth muscle cells and is prevented by glucose transport and metabolism	scientific article published on 27 August 2002

Statements

scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

Enhanced glycogen synthase kinase-3beta activity mediates hypoxia-induced apoptosis of vascular smooth muscle cells and is prevented by glucose transport and metabolism (English)

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

0 references

apoptotic process

1 reference

based on heuristic

inferred from title

glycogen metabolic process

1 reference

based on heuristic

inferred from title

author name string

Robert D Loberg

1

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

Eileen Vesely

2

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

Frank C Brosius

3

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

language of work or name

0 references

publication date

27 August 2002

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

Journal of Biological Chemistry

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Europe PubMed Central

PubMed publication ID

30 November 2017

277

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

44

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

41667-41673

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

Apoptosis signaling

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M206405200

21 January 2018

Glucose uptake and glycolysis reduce hypoxia-induced apoptosis in cultured neonatal rat cardiac myocytes

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M206405200

21 January 2018

Hypoxia induces apoptosis via two independent pathways in Jurkat cells: differential regulation by glucose

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M206405200

21 January 2018

ARC inhibits cytochrome c release from mitochondria and protects against hypoxia-induced apoptosis in heart-derived H9c2 cells

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M206405200

21 January 2018

Proapoptotic stimuli induce nuclear accumulation of glycogen synthase kinase-3 beta

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M206405200

21 January 2018

Judging a protein by more than its name: GSK-3.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M206405200

21 January 2018

The renaissance of GSK3

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M206405200

21 January 2018

Glycogen synthase kinase3 beta phosphorylates serine 33 of p53 and activates p53's transcriptional activity

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M206405200

21 January 2018

Caspase-3 activation induced by inhibition of mitochondrial complex I is facilitated by glycogen synthase kinase-3beta and attenuated by lithium

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M206405200

21 January 2018

Growth factor withdrawal from primary human erythroid progenitors induces apoptosis through a pathway involving glycogen synthase kinase-3 and Bax.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M206405200

21 January 2018

The protein kinase PKB/Akt regulates cell survival and apoptosis by inhibiting Bax conformational change

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M206405200

21 January 2018

Cyclic AMP promotes neuronal survival by phosphorylation of glycogen synthase kinase 3beta.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M206405200

21 January 2018

In the absence of extrinsic signals, nutrient utilization by lymphocytes is insufficient to maintain either cell size or viability

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M206405200

21 January 2018

Akt and Bcl-xL promote growth factor-independent survival through distinct effects on mitochondrial physiology

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M206405200

21 January 2018

Upregulation of glucose metabolism during intimal lesion formation is coupled to the inhibition of vascular smooth muscle cell apoptosis. Role of GSK3beta

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M206405200

21 January 2018

Glycogen synthase kinase-3beta facilitates staurosporine- and heat shock-induced apoptosis. Protection by lithium

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M206405200

21 January 2018

Glucagon-like peptide-2 receptor activation engages bad and glycogen synthase kinase-3 in a protein kinase A-dependent manner and prevents apoptosis following inhibition of phosphatidylinositol 3-kinase.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M206405200

21 January 2018

Role of glycogen synthase kinase-3 in the phosphatidylinositol 3-Kinase/Akt cell survival pathway

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M206405200

21 January 2018

Role of translation initiation factor 2B in control of cell survival by the phosphatidylinositol 3-kinase/Akt/glycogen synthase kinase 3beta signaling pathway

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M206405200

21 January 2018

Requirement for glycogen synthase kinase-3beta in cell survival and NF-kappaB activation

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M206405200

21 January 2018

Multiple signaling pathways mediate LIF-induced skeletal muscle satellite cell proliferation

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M206405200

21 January 2018

Decreased glucose transporter expression triggers BAX-dependent apoptosis in the murine blastocyst

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M206405200

21 January 2018

Identifiers

10.1074/JBC.M206405200

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

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