Expression of Nav1.7 in DRG neurons extends from peripheral terminals in the skin to central preterminal branches and terminals in the dorsal horn (Q41901986)

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scientific article published on 7 November 2012

Language	Label	Description	Also known as
English	Expression of Nav1.7 in DRG neurons extends from peripheral terminals in the skin to central preterminal branches and terminals in the dorsal horn	scientific article published on 7 November 2012

Statements

scholarly article

1 reference

Europe PubMed Central

PMC publication ID

13 October 2017

Expression of Nav1.7 in DRG neurons extends from peripheral terminals in the skin to central preterminal branches and terminals in the dorsal horn (English)

1 reference

Europe PubMed Central

PMC publication ID

13 October 2017

author name string

Joel A Black

1

1 reference

Europe PubMed Central

PMC publication ID

13 October 2017

Noémie Frézel

2

1 reference

Europe PubMed Central

PMC publication ID

13 October 2017

Sulayman D Dib-Hajj

3

1 reference

Europe PubMed Central

PMC publication ID

13 October 2017

Stephen G Waxman

4

1 reference

Europe PubMed Central

PMC publication ID

13 October 2017

publication date

7 November 2012

1 reference

Europe PubMed Central

PMC publication ID

13 October 2017

1 reference

Europe PubMed Central

PMC publication ID

13 October 2017

8

1 reference

Europe PubMed Central

PMC publication ID

13 October 2017

82

1 reference

Europe PubMed Central

PMC publication ID

13 October 2017

Distinct Nav1.7-dependent pain sensations require different sets of sensory and sympathetic neurons

1 reference

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Reduction of voltage gated sodium channel protein in DRG by vector mediated miRNA reduces pain in rats with painful diabetic neuropathy

1 reference

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Intra- and interfamily phenotypic diversity in pain syndromes associated with a gain-of-function variant of NaV1.7.

1 reference

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The roles of sodium channels in nociception: implications for mechanisms of neuropathic pain

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Gain of function NaV1.7 mutations in idiopathic small fiber neuropathy

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Nav1.7 is the predominant sodium channel in rodent olfactory sensory neurons

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Loss-of-function mutations in sodium channel Nav1.7 cause anosmia.

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Sodium-calcium exchanger and multiple sodium channel isoforms in intra-epidermal nerve terminals.

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Mutations at opposite ends of the DIII/S4-S5 linker of sodium channel Na V 1.7 produce distinct pain disorders

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Sodium channels in normal and pathological pain

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Targeting ion channels for drug discovery.

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ERK1/2 mitogen-activated protein kinase phosphorylates sodium channel Na(v)1.7 and alters its gating properties

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Voltage-gated sodium channels: therapeutic targets for pain

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Sensory neuron voltage-gated sodium channels as analgesic drug targets

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Paroxysmal extreme pain disorder M1627K mutation in human Nav1.7 renders DRG neurons hyperexcitable

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Comparative study of the distribution of the alpha-subunits of voltage-gated sodium channels in normal and axotomized rat dorsal root ganglion neurons

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Continuous delta-opioid receptor activation reduces neuronal voltage-gated sodium channel (NaV1.7) levels through activation of protein kinase C in painful diabetic neuropathy

1 reference

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From genes to pain: Na v 1.7 and human pain disorders

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Changes in the expression of NaV1.7, NaV1.8 and NaV1.9 in a distinct population of dorsal root ganglia innervating the rat knee joint in a model of chronic inflammatory joint pain

1 reference

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Distribution of the voltage-gated sodium channel Na(v)1.7 in the rat: expression in the autonomic and endocrine systems.

1 reference

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A stop codon mutation in SCN9A causes lack of pain sensation

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Loss-of-function mutations in the Nav1.7 gene underlie congenital indifference to pain in multiple human populations

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5 June 2018

An SCN9A channelopathy causes congenital inability to experience pain

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5 June 2018

Multiple sodium channels and their roles in electrogenesis within dorsal root ganglion neurons

1 reference

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SCN9A mutations in paroxysmal extreme pain disorder: allelic variants underlie distinct channel defects and phenotypes

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A single sodium channel mutation produces hyper- or hypoexcitability in different types of neurons

1 reference

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Sporadic onset of erythermalgia: a gain-of-function mutation in Nav1.7.

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International Union of Pharmacology. XLVII. Nomenclature and structure-function relationships of voltage-gated sodium channels

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Contactin regulates the current density and axonal expression of tetrodotoxin-resistant but not tetrodotoxin-sensitive sodium channels in DRG neurons

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Gain-of-function mutation in Nav1.7 in familial erythromelalgia induces bursting of sensory neurons

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SCN9A mutations define primary erythermalgia as a neuropathic disorder of voltage gated sodium channels

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Nociceptor-specific gene deletion reveals a major role for Nav1.7 (PN1) in acute and inflammatory pain

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Early painful diabetic neuropathy is associated with differential changes in tetrodotoxin-sensitive and -resistant sodium channels in dorsal root ganglion neurons in the rat.

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Mutations in SCN9A, encoding a sodium channel alpha subunit, in patients with primary erythermalgia

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Distinct repriming and closed-state inactivation kinetics of Nav1.6 and Nav1.7 sodium channels in mouse spinal sensory neurons

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Accumulation of PN1 and PN3 sodium channels in painful human neuroma-evidence from immunocytochemistry

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Slow closed-state inactivation: a novel mechanism underlying ramp currents in cells expressing the hNE/PN1 sodium channel.

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Identification of PN1, a predominant voltage-dependent sodium channel expressed principally in peripheral neurons

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Structure and functional expression of a new member of the tetrodotoxin-sensitive voltage-activated sodium channel family from human neuroendocrine cells

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Electrical properties of rat dorsal root ganglion neurones with different peripheral nerve conduction velocities.

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NeuN, a neuronal specific nuclear protein in vertebrates

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Multiple sodium channel isoforms and mitogen-activated protein kinases are present in painful human neuromas.

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Sensory and electrophysiological properties of guinea-pig sensory neurones expressing Nav 1.7 (PN1) Na+ channel alpha subunit protein

1 reference

https://pubmed.ncbi.nlm.nih.gov/23134641

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Microelectrode recordings from transected nerves in amputees with phantom limb pain

1 reference

https://pubmed.ncbi.nlm.nih.gov/23134641

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

NF-L and peripherin immunoreactivities define distinct classes of rat sensory ganglion cells

1 reference

https://pubmed.ncbi.nlm.nih.gov/23134641

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Na(v)1.7 sodium channel expression in human lingual nerve neuromas

1 reference

https://pubmed.ncbi.nlm.nih.gov/23134641

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Nav1.7-related small fiber neuropathy: impaired slow-inactivation and DRG neuron hyperexcitability

1 reference

https://pubmed.ncbi.nlm.nih.gov/23134641

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1186/1744-8069-8-82

1 reference

Europe PubMed Central

PMC publication ID

13 October 2017

Dimensions Publication ID

0 references

PMC publication ID

1 reference

Europe PubMed Central

PMC publication ID

13 October 2017

PubMed publication ID

1 reference

Europe PubMed Central

PMC publication ID

13 October 2017

ResearchGate publication ID

0 references

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