Dominant negative Met reduces tumorigenicity-metastasis and increases tubule formation in mammary cells (Q40877285)

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English	Dominant negative Met reduces tumorigenicity-metastasis and increases tubule formation in mammary cells	scientific article

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scholarly article

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Europe PubMed Central

PubMed publication ID

24 September 2017

Dominant negative Met reduces tumorigenicity-metastasis and increases tubule formation in mammary cells (English)

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Europe PubMed Central

PubMed publication ID

24 September 2017

author name string

Firon M

1

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24 September 2017

Shaharabany M

2

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Europe PubMed Central

PubMed publication ID

24 September 2017

Altstock RT

3

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Europe PubMed Central

PubMed publication ID

24 September 2017

Horev J

4

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Europe PubMed Central

PubMed publication ID

24 September 2017

Abramovici A

5

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

Resau JH

6

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Europe PubMed Central

PubMed publication ID

24 September 2017

Vande Woude GF

7

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Europe PubMed Central

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24 September 2017

Tsarfaty I

8

1 reference

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24 September 2017

language of work or name

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publication date

1 May 2000

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Europe PubMed Central

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24 September 2017

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Europe PubMed Central

PubMed publication ID

24 September 2017

19

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24 September 2017

20

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Europe PubMed Central

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24 September 2017

2386-2397

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Europe PubMed Central

PubMed publication ID

24 September 2017

Expression of a dominant negative mutant of the FGF receptor disrupts mesoderm formation in Xenopus embryos

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Expression of the C-Met/HGF receptor in human breast carcinoma: Correlation with tumor progression

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Induction of epithelial tubules by growth factor HGF depends on the STAT pathway

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Hepatocyte growth factor is a potent angiogenic factor which stimulates endothelial cell motility and growth

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The Met-HGF/SF autocrine signaling mechanism is involved in sarcomagenesis

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Overexpression of the Met/HGF receptor in ovarian cancer.

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Growth Factor Synthesis and Human Breast Cancer Progression

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Expression of c-met is a strong independent prognostic factor in breast carcinoma

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Transfer of motogenic and invasive response to scatter factor/hepatocyte growth factor by transfection of human MET protooncogene

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Characterization of the TPR-MET oncogene p65 and the MET protooncogene p140 protein-tyrosine kinases

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The Rous sarcoma virus long terminal repeat is a strong promoter when introduced into a variety of eukaryotic cells by DNA-mediated transfection

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sprouty Encodes a Novel Antagonist of FGF Signaling that Patterns Apical Branching of the Drosophila Airways

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Enhanced tumorigenicity and invasion-metastasis by hepatocyte growth factor/scatter factor-met signalling in human cells concomitant with induction of the urokinase proteolysis network

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Activating mutations for the Met tyrosine kinase receptor in human cancer

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Expression of scatter factor and c-met receptor in benign and malignant breast tissue

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Nonparametric Estimation from Incomplete Observations

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Transgenic expression of tpr-met oncogene leads to development of mammary hyperplasia and tumors

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Expression and loss of heterozygosity of c-met proto-oncogene in primary breast cancer

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Molecular cloning and expression of human hepatocyte growth factor

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Expression of the c-Met/HGF receptor in human melanocytic neoplasms: demonstration of the relationship to malignant melanoma tumour progression

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Modulation of hepatocyte growth factor and c-met in the rat mammary gland during pregnancy, lactation, and involution

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A multifunctional docking site mediates signaling and transformation by the hepatocyte growth factor/scatter factor receptor family

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Dimerization mediated through a leucine zipper activates the oncogenic potential of the met receptor tyrosine kinase

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Tumorigenicity of the met proto-oncogene and the gene for hepatocyte growth factor

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Invasiveness and metastasis of NIH 3T3 cells induced by Met-hepatocyte growth factor/scatter factor autocrine stimulation

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Smooth muscle releases an epithelial cell scatter factor which binds to heparin

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Scatter factor modulates the metastatic phenotype of the EMT6 mouse mammary tumor

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Scatter factor/hepatocyte growth factor is essential for liver development

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Germline and somatic mutations in the tyrosine kinase domain of the MET proto-oncogene in papillary renal carcinomas

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Scatter factor is a fibroblast-derived modulator of epithelial cell mobility

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Mutation of juxtamembrane tyrosine residue 1001 suppresses loss-of-function mutations of the met receptor in epithelial cells.

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10.1038/SJ.ONC.1203557

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Europe PubMed Central

PubMed publication ID

24 September 2017

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PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

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